gms | German Medical Science

84th Annual Meeting of the German Society of Oto-Rhino-Laryngology, Head and Neck Surgery

German Society of Oto-Rhino-Laryngology, Head and Neck Surgery

08.05. - 12.05.2013, Nürnberg

Patency of the cochlear aqueduct in Menière’s disease

Meeting Abstract

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  • corresponding author presenting/speaker Jonas Jae-Hyun Park - Department of Otolaryngology and Head and Neck Surgery, University Hospital of Aachen, Aachen, Germany
  • author Anmin Shen - Department of Otolaryngology and Head and Neck Surgery, University Hospital of Aachen, Aachen, Germany
  • author Martin Westhofen - Department of Otolaryngology and Head and Neck Surgery, University Hospital of Aachen, Aachen, Germany

German Society of Oto-Rhino-Laryngology, Head and Neck Surgery. 84th Annual Meeting of the German Society of Oto-Rhino-Laryngology, Head and Neck Surgery. Nürnberg, 08.-12.05.2013. Düsseldorf: German Medical Science GMS Publishing House; 2013. Doc13hno23

doi: 10.3205/13hno23, urn:nbn:de:0183-13hno236

Published: July 30, 2013

© 2013 Park et al.
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Outline

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Introduction: The cochlear aqueduct (CA) plays an important role in the hydrostatic inner ear pressure regulation. Its function can be impaired in inner ear pressure disorders such as in Menière’s disease. Due to its narrow diameter the patency of CA in patients with Menière’s disease is not known. The aim of this study was to compare the patency of CA in patients with Menière’s disease to healthy persons by radiological examinations.

Patients and methods: High resolution computed tomography (HRCT) and high resolution magnetic resonance tomography (HRMRT) of temporal bones of 73 patients with Menière’s disease and of 30 healthy persons were analyzed retrospectively.

Results: Bony diameters in CA in HRCT did not show any differences between both groups. Fluid length was reduced in HRMRT in patients with Menière’s disease compared to healthy persons.

Conclusion: Reduced CA fluid length in HRMRT in Menière’s disease patients might be due to increased fibrous tissue in CA leading to impaired patency of CA.