gms | German Medical Science

Deutscher Kongress für Orthopädie und Unfallchirurgie (DKOU 2017)

24.10. - 27.10.2017, Berlin

Effect of capsule repair on rotational and varus stability in PLRI reconstruction

Meeting Abstract

  • presenting/speaker Michael Künzler - Inselspital Bern, Department of Orthopaedics and Traumatology, Bern, Switzerland
  • Masaki Akeda - University of California, Irvine, Orthopaedics Biomechanics Laboratory, Long Beach, United States
  • Hansel Ihn - University of California, Irvine, Orthopaedics Biomechanics Laboratory, Long Beach, United States
  • Michelle McGarry - University of California, Irvine, Orthopaedics Biomechanics Laboratory, Long Beach, United States
  • Matthias Zumstein - Inselspital Bern, Department of Orthopaedics and Traumatology, Bern, Switzerland
  • Thay Q Lee - University of California, Irvine, Orthopaedics Biomechanics Laboratory, Long Beach, United States

Deutscher Kongress für Orthopädie und Unfallchirurgie (DKOU 2017). Berlin, 24.-27.10.2017. Düsseldorf: German Medical Science GMS Publishing House; 2017. DocGR12-256

doi: 10.3205/17dkou483, urn:nbn:de:0183-17dkou4833

Veröffentlicht: 23. Oktober 2017

© 2017 Künzler et al.
Dieser Artikel ist ein Open-Access-Artikel und steht unter den Lizenzbedingungen der Creative Commons Attribution 4.0 License (Namensnennung). Lizenz-Angaben siehe http://creativecommons.org/licenses/by/4.0/.


Gliederung

Text

Objectives: Insufficiency of the lateral collateral ligamentous complex causes posterolateral rotatory instability (PLRI). During reconstruction surgery the joint capsule is repaired, but its biomechanical influence on elbow stability has not been described. We hypothesized that capsular repair reduces ROM and varus angle after reconstruction of the lateral collateral complex.

Methods: Six fresh frozen cadaveric elbow specimens were used. Varus laxity in supination, pronation and neutral forearm rotation with 1 Nm load and forearm rotaitonal range of motion (ROM) with 0.3 Nm torque were measured using a Microscribe 3DLX digitizing system (Revware Inc, Raleigh, NC). Each specimen was tested under four different conditions: Intact, Complete Tear with LUCL, RCL and capsule tear, LUCL/RCL reconstruction + capsule repair and LUCL/RCL reconstruction only. Reconstruction was performed according to the docking technique (Jones, JSES, 2013) and the capsule was repaired with mattress sutures. Each condition was tested in 30°, 60° and 90° elbow flexion. A two-way ANOVA with Tukey's post-hoc test was used to detect statistical differences between the conditions.

Results and Conclusion: Total ROM of the forearm significantly increased in all flexion angles from intact to Complete tear (p< 0.001). ROM was restored to normal in 30° and 60° elbow flexion in both reconstruction conditions (p >0.05). LUCL/RCL Reconstruction + capsule repair in 90° elbow flexion was associated with a significantly lower ROM compared to intact (p=0.0003) and reconstruction without capsule repair (p=0.015). Varus angle increased significantly from intact to complete tear (p< 0.0001) and restored to normal in both reconstruction conditions (p >0.05) in 30° and 60° elbow flexion. In contrast varus angle was significantly lower in 90° elbow flexion in both reconstruction conditions compared to intact (both p<0.0001).

Reconstruction of the lateral collateral complex restores elbow stability, ROM and varus laxity independent of capsular repair. Over tightening of the elbow joint occurred in 90° elbow flexion, which was aggravated by capsular repair. This overtightening causes stress to the transplant and might cause failure of the reconstruction. Therefore to avoid recurrence of PLRI, postoperative rehabilitation should avoid high degrees of elbow flexion to protect the reconstruction from wearing out.