27. Wissenschaftlicher Kongress der Deutschen Hochdruckliga
Deutsche Liga zur Bekämpfung des hohen Blutdrucks – Deutsche Hypertonie Gesellschaft e. V.
26. bis 29.11.2003, Bonn
Postersession
Genetische Aspekte
Meeting Abstract
(03hochP34)
[Volltext]
The G(s)alpha gene as a predictor of successful weight loss and cardiovascular side effects with sibutramine
Frey UH, Hauner H, Bachmann HS, Meier M, Siffert W[Volltext]
Meeting Abstract
(03hochP35)
[Volltext]
Altered gene expression proflie in rats with and without terminal heart failure
Dechend R, Al-Saadi N, Kirsch T, Wellner M, Fiebeler A, Haller H, Luft FC, Müller DN[Volltext]
Meeting Abstract
(03hochP36)
[Volltext]
Genexpression of Nitric Oxide Synthase Isoforms in the HPA Axis of STZ-Induced Diabetic Rats
Stark E, Qadri F, Häuser W, Jöhren O, Dendorfer A, Dominiak P[Volltext]
Meeting Abstract
(03hochP37)
[Volltext]
Different genetic loci are involved in the development of salt-independent early onset albuminuria and in salt-dependent progressive renal damage
Siegel AK, Mehr Poyan A, Planert M, Schulz A, Kreutz R[Volltext]
Meeting Abstract
(03hochP38)
[Volltext]
Vascular Response to Insulin is Determined by the C825T Polymorphism of the G Protein β3 Subunit
Mitchell A, Pace M, Nürnberger J, Siffert W, Wenzel RR, Philipp T, Schäfers RF[Volltext]
Meeting Abstract
(03hochP39)
[Volltext]
The G protein-coupled receptor Mas is a physiological antagonist of the angiotensin II AT1 receptor
Walther T, Milligan G, Christopoulos A, Sanchez-Ferrer C, Heringer-Walther S, Sexton P, Schultheiss H, Kostenis E[Volltext]
Meeting Abstract
(03hochP40)
[Volltext]
Systematic genetic mapping analysis of P2-Purinoceptor-Loci in the rat genome
Viviano E, Siegel AK, Schulz A, Kreutz R[Volltext]
Meeting Abstract
(03hochP41)
[Volltext]
Deficiency for the G protein-coupled receptor Mas prevents inversion of circadian rhythmus by Angiotensin II
Heringer-Walther S, Schultheiss HP, Walther T[Volltext]
Meeting Abstract
(03hochP42)
[Volltext]
Transgenic human tissue kallikrein rats are not protected after induction of myocardial infarction
Koch M, Westermann D, Wendorf M, Dendorfer A, Spillmann F, Bader M, Walther T, Schultheiss HP, Tschöpe C[Volltext]
